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PreclinicalResearch compoundLimited evidence

ASP2905

Selective Kv12.2 (KCNH3) potassium channel inhibitor, attention-enhancing candidate, preclinical data only.

NootropicMemoryMemoryAttentionAcetylcholineDopaminenoo.affects.attention

Pharmacology

ClassMemory · Attention
Primary targetKv12.2 (KCNH3) voltage-gated potassium channel inhibition
Targets3 receptor targets
Half-lifeNo published human pharmacokinetic data
OnsetNo human data
EvidenceLimited evidence
Affected systemsAcetylcholineDopaminenoo.affects.attention

Contents

WHAT IS ASP2905?

Detailed overview

ASP2905 is an experimental compound developed by Astellas that acts as a potent and selective inhibitor of the Kv12.2 (KCNH3-encoded) voltage-gated potassium channel. This channel is concentrated in the forebrain: its overexpression produces cognitive deficits in rodents, whereas its knockout improves attention and learning, so blocking the channel increases excitatory neuronal activity in cognitive circuits. In mouse models ASP2905 reversed memory deficits induced by MK-801 and scopolamine, improved learning in aged rats, and showed antipsychotic-like activity as well as attention-enhancing effects in an ADHD model; in rat microdialysis it increased dopamine and acetylcholine efflux in the medial prefrontal cortex. There is no human clinical data: efficacy and safety in humans are unstudied and the compound exists only as a preclinical research chemical.

Mechanism

Kv12.2 (KCNH3) potassium channel inhibition

Evidence

Preclinical only (no human data)

Legal status

Unapproved research chemical

Receptor profile

  • Kv12.2 / KCNH3 potassium channelStrong
  • Prefrontal dopamine efflux (indirect)Moderate
  • Prefrontal acetylcholine efflux (indirect)Moderate

Safety

Side effects, stop signs, contraindications

Side effects · 5

  • No human safety data: only preclinical (mouse and rat) studies exist; the compound has never been tested in humans
  • Increased neuronal excitability may theoretically cause restlessness, anxiety or sleep disturbance
  • A psychoactive (behavior-altering) effect was demonstrated in mice, indicating dose-dependent behavioral change
  • Theoretical seizure risk, since Kv12.2 inhibition raises excitatory neuronal activity in the forebrain
  • Unknown long-term safety and missing toxicity profile in humans

Contraindications · 4

  • Pregnancy and breastfeeding: safety not established, avoid
  • Epilepsy or lowered seizure threshold: increased forebrain excitability may theoretically raise seizure risk
  • Avoid combining with other stimulant / dopaminergic agents due to additive overstimulation
  • Not an approved medicine; experimental research chemical, not recommended without clinical supervision

Related Nootropics

Same therapeutic category

Studies

Related research and clinical findings

FAQ

FAQ

Selective Kv12.2 (KCNH3) potassium channel inhibitor, attention-enhancing candidate, preclinical data only.

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Structure & chemistry

TypeNootropic
FormulaC20H17FN8
UpdatedJuly 10, 2026
MolekulaX Editorial Team·Source-verified · PubMed · FDA · EMA
Updated: July 10, 2026

The information here is strictly for educational and scientific purposes. It does not replace medical advice or clinical consultation, and it does not encourage illegal substance or pharmaceutical use. Data is sourced. When in doubt, consult your doctor.