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PreclinicalResearch compoundLimited evidence

Eutropoflavin

Synthetic flavone TrkB agonist, a more potent and stable derivative of tropoflavin (7,8-DHF). Preclinical data only.

NootropicLongevityBDNFNeuroprotectionBDNFnoo.affects.neuroprotectionnoo.affects.mood

Pharmacology

ClassBDNF · Neuroprotection
Primary targetSelective TrkB receptor agonism (BDNF mimetic)
Targets3 receptor targets
Half-lifeNo human pharmacokinetic data (in rodents the effect peaks at ~4 hours and partially decays around 8-16 hours)
OnsetNo human data (in rodents TrkB phosphorylation peaks within hours)
EvidenceLimited evidence
Affected systemsBDNFnoo.affects.neuroprotectionnoo.affects.mood

Contents

WHAT IS EUTROPOFLAVIN?

Detailed overview

Eutropoflavin (4'-dimethylamino-7,8-dihydroxyflavone) is a synthetic flavone that acts as a selective small-molecule agonist of tropomyosin receptor kinase B (TrkB), the receptor for BDNF (brain-derived neurotrophic factor), so it mimics BDNF signaling. Compared with tropoflavin (7,8-dihydroxyflavone), it carries a dimethylamino group at the 4' position, which raises its lipophilicity, improves blood-brain-barrier penetration and metabolic stability, making it more potent and longer-lasting than the parent compound. In rodent models it produced neuroprotective, neurogenic and antidepressant-like effects, including hippocampal neurogenesis. There are no human clinical trials: safety and efficacy in humans are unstudied and the compound has never been approved for human use, so it exists only as an experimental research chemical.

Mechanism

Selective TrkB agonist (BDNF mimetic)

Evidence

Preclinical only (no human clinical data)

Legal status

Unapproved research chemical

Receptor profile

  • TrkB receptor (BDNF receptor)Strong
  • BDNF downstream signaling (ERK / CREB / Akt)Strong
  • Hippocampal neurogenesis and synaptic plasticityModerate

Safety

Side effects, stop signs, contraindications

Side effects · 5

  • No human safety data: only preclinical (rodent) studies exist; the compound has not been tested in humans in controlled clinical trials
  • Theoretical risks of sustained TrkB activation: excessive neurotrophic signaling could in principle carry unfavorable cell-growth effects
  • Possible gastrointestinal discomfort or headache at higher doses, typical of flavonoids
  • Unknown long-term safety and incomplete toxicity profile in humans
  • Unregulated quality and purity: as a research-chemical product, dose and contamination can vary

Contraindications · 3

  • Pregnancy and breastfeeding: safety not established, avoid
  • Active or prior malignancy: TrkB signaling is overactive in some tumor types, so heightened caution is warranted
  • Not an approved medicine; experimental compound, not recommended without clinical supervision

Related Nootropics

Same therapeutic category

Studies

Related research and clinical findings

FAQ

FAQ

Synthetic flavone TrkB agonist, a more potent and stable derivative of tropoflavin (7,8-DHF). Preclinical data only.

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Structure & chemistry

TypeNootropic
FormulaC17H15NO4
UpdatedJuly 10, 2026
MolekulaX Editorial Team·Source-verified · PubMed · FDA · EMA
Updated: July 10, 2026

The information here is strictly for educational and scientific purposes. It does not replace medical advice or clinical consultation, and it does not encourage illegal substance or pharmaceutical use. Data is sourced. When in doubt, consult your doctor.