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PreclinicalResearch compoundLimited evidence

PPAP

Deprenyl-derived catecholaminergic activity enhancer (CAE) with no MAO inhibition; preclinical data only.

NootropicDopaminergicDopaminergicNeuroprotectionDopamineNorepinephrine

Pharmacology

ClassDopaminergic · Neuroprotection
Primary targetEnhancement of impulse-coupled catecholamine release (CAE)
Targets3 receptor targets
Half-lifeNo human pharmacokinetic data
OnsetNo human data
EvidenceLimited evidence
Affected systemsDopamineNorepinephrine

Contents

WHAT IS PPAP?

Detailed overview

PPAP (1-phenyl-2-propylaminopentane) is a structural analogue of deprenyl (selegiline), developed by József Knoll and colleagues at the Department of Pharmacology, Semmelweis University in Budapest in the late 1980s. It is the prototype of the so-called catecholaminergic activity enhancers (CAE): unlike amphetamine it does not force transmitter release, and unlike selegiline it does not inhibit MAO. Instead it strengthens the coupling between a neuron's action potential and its transmitter release, so dopaminergic and noradrenergic neurons discharge more catecholamine when stimulated. In contrast to selegiline it is not metabolized to amphetamine. More recent data indicate it is also a catecholamine reuptake inhibitor (DAT IC50 ~57.5 nM). It has never been tested in humans and exists only as an experimental research chemical.

Mechanism

Catecholaminergic activity enhancer (CAE), non-MAO-inhibiting

Evidence

Preclinical only (no human data)

Legal status

Unapproved; controlled in several countries

Receptor profile

  • Catecholaminergic activity enhancement (CAE)Strong
  • Catecholamine reuptake (DAT/NET)Moderate
  • Neuronal survival signalingModerate

Safety

Side effects, stop signs, contraindications

Side effects · 5

  • No human safety data: the compound has never been tested in humans, all evidence comes from rodent studies
  • Possible overstimulation: anxiety, restlessness, insomnia from the heightened catecholaminergic tone
  • Increased heart rate and elevated blood pressure, as expected from sympathomimetic/stimulant agents
  • Theoretical addiction and abuse potential via dopaminergic reinforcement, though lower than amphetamine
  • Unknown long-term safety and incomplete toxicity profile in humans

Contraindications · 5

  • Pregnancy and breastfeeding: safety not established, avoid
  • Cardiovascular disease, arrhythmia, uncontrolled hypertension: the sympathomimetic effect may worsen them
  • Anxiety disorders, panic disorder: the stimulant effect may aggravate symptoms
  • Avoid combining with MAO inhibitors and other serotonergic/adrenergic agents due to catecholaminergic overload risk
  • Not an approved medicine; experimental research chemical, not recommended without clinical supervision

Related Nootropics

Same therapeutic category

Studies

Related research and clinical findings

FAQ

FAQ

Deprenyl-derived catecholaminergic activity enhancer (CAE) with no MAO inhibition; preclinical data only.

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Structure & chemistry

TypeNootropic
FormulaC14H23N
UpdatedJuly 10, 2026
MolekulaX Editorial Team·Source-verified · PubMed · FDA · EMA
Updated: July 10, 2026

The information here is strictly for educational and scientific purposes. It does not replace medical advice or clinical consultation, and it does not encourage illegal substance or pharmaceutical use. Data is sourced. When in doubt, consult your doctor.